ඇන්ටි බොඩි

[lodbrok]

කොරෝනා එක පාරක් හැදුන කෙනෙකුට තව පාරක් හැදෙනවද කියල අහන ප්‍රශ්නෙට දැනට මම දැකපු හොඳම උත්තරේ තමයි මේ තියෙන්නෙ.

ඇන්ටිබොඩීස් වලට වයිරස් එක මතක තියෙන්නෙ බොහොම පොඩි කාලයයි.

ඒ හින්ද ළඟ ළඟ හැදෙන්නෙ නැති උනත් අවුරුද්දකට සැරයක්වත් ආපහු හැදෙයි.

හැබැයි නිතරම වයිරස් එකට නිරාවරණය වෙන අයගෙ ඇන්ටිබොඩි දිගටම ෆයිට් එක දෙන සීන් එකක් තියෙන්නෙ.


The Imperial College London team found the number of people testing positive for antibodies has fallen by 26% between June and September.

They say immunity appears to be fading and there is a risk of catching the virus multiple times.

In the first round of testing, at the end of June and the beginning of July, about 60 in 1,000 people had detectable antibodies.

But in the latest set of tests, in September, only 44 per 1,000 people were positive.

https://www.bbc.co.uk/news/health-54696873

 

[Mohowk]

 

[dilshan87]

Ammata siri. E kiyanne hadan berenawa kiyanne boru wadak. Jeewitha kalema vaccine gagaha inna wei.

 

[NRTG]

BUMP BRO....

 

[Randir]

Influenza injection ekat yearly Hari once in 2 years Hari gahanna one bn. Okat ehema wei

 

[Gonawala_GUNE]

ඇන්ටිගේ බොඩි

 

[MySelf99]

Aunty body

 

[kinkon]

බම්ප්

 

[deadpool84]

nodakin hoda aunty bodiyak balanna awa.

 

[imhotep]

කොරෝනා එක පාරක් හැදුන කෙනෙකුට තව පාරක් හැදෙනවද කියල අහන ප්‍රශ්නෙට දැනට මම දැකපු හොඳම උත්තරේ තමයි මේ තියෙන්නෙ.

ඇන්ටිබොඩීස් වලට වයිරස් එක මතක තියෙන්නෙ බොහොම පොඩි කාලයයි.

ඒ හින්ද ළඟ ළඟ හැදෙන්නෙ නැති උනත් අවුරුද්දකට සැරයක්වත් ආපහු හැදෙයි.

හැබැයි නිතරම වයිරස් එකට නිරාවරණය වෙන අයගෙ ඇන්ටිබොඩි දිගටම ෆයිට් එක දෙන සීන් එකක් තියෙන්නෙ.


The Imperial College London team found the number of people testing positive for antibodies has fallen by 26% between June and September.

They say immunity appears to be fading and there is a risk of catching the virus multiple times.

In the first round of testing, at the end of June and the beginning of July, about 60 in 1,000 people had detectable antibodies.

But in the latest set of tests, in September, only 44 per 1,000 people were positive.

https://www.bbc.co.uk/news/health-54696873

I posted the possible explanation for this on last Sunday.. Only a few people bumped it. (but sadly people keep on replying to posts by mentally retarded )

Here it is....

https://elakiri.com/threads/low-quality-of-antibody-production-in-serious-covid-19-cases.1958678/

Copied below......

For anyone interested....

Possible explanation of why the low quality of antibodies found in those infected with Covid, and the limited B-cell memory has been published by a team of researchers led by Prof Shiv Pillai (Professor of Medicine and Health Sciences at Harvard, Director of Harvard immunology)
They attribute it to the loss of germinal centers in Covid-19 patients. During a viral infection, foreign antigens carried to the secondary lymphoid organs are recognized by the B-cells and this mechanism takes place in a germinal center. The germinal center is a specialised microstructure where production of long-lived memory B cells (which provides protection from reinfection) takes place.
It is suspected that the Cytokine storm in severe Covid-19 cases is partly responsible for the loss of these germinal centers. It is highly unlikely that any of the vaccines developed will cause the loss of germinal centers as the vaccines do not have the live virus itself.

Another unknown issue at this stage is why some people develop into serious Covid-19 and why some do not. Immunologists suspect that this has something to do with the lymphocyte function and currently trying to to unravel the reasons.

Here's an extract from their publication. It's not written to be easily understood by an average person but below is a simple summary which is important.

"Absence of Germinal Centers and Loss of Bcl-6+ Germinal Center B Cells but Preservation of AID+ B Cells in Lymph Nodes Early in COVID-19 Disease. We have used a human-tissue-imaging platform with quantitative high-resolution automated slide-scanning microscopy, exploiting both regular and confocal approaches and multispectral imaging, in order to interrogate human lymphoid and non-lymphoid organs at the single-cell level. These approaches crucially preserve architecture over broad swaths of tissue. Thoracic lymph nodes in severely ill COVID-19 patients who succumbed in less than 8 days after admission, less than 10 days from the onset of respiratory symptoms displayed a lack of germinal centers, and these were also absent in those who succumbed later, 15–36 days after admission."

"These data suggest an underlying basis for the lower quality and lack of durability of humoral immune responses observed during natural infection with SARS-CoV-2 and have significant implications for expectations of herd immunity. "

 

[PHBhagya]

බම්ප්

 

[pavithra_uk]

lankawe ekekta 3 parak hadila thibba within 4 months

 

[navinnuwan]

I posted the possible explanation for this on last Sunday.. Only a few people bumped it. (but sadly people keep on replying to posts by mentally retarded )

Here it is....

https://elakiri.com/threads/low-quality-of-antibody-production-in-serious-covid-19-cases.1958678/

Copied below......

For anyone interested....

Possible explanation of why the low quality of antibodies found in those infected with Covid, and the limited B-cell memory has been published by a team of researchers led by Prof Shiv Pillai (Professor of Medicine and Health Sciences at Harvard, Director of Harvard immunology)
They attribute it to the loss of germinal centers in Covid-19 patients. During a viral infection, foreign antigens carried to the secondary lymphoid organs are recognized by the B-cells and this mechanism takes place in a germinal center. The germinal center is a specialised microstructure where production of long-lived memory B cells (which provides protection from reinfection) takes place.
It is suspected that the Cytokine storm in severe Covid-19 cases is partly responsible for the loss of these germinal centers. It is highly unlikely that any of the vaccines developed will cause the loss of germinal centers as the vaccines do not have the live virus itself.

Another unknown issue at this stage is why some people develop into serious Covid-19 and why some do not. Immunologists suspect that this has something to do with the lymphocyte function and currently trying to to unravel the reasons.

Here's an extract from their publication. It's not written to be easily understood by an average person but below is a simple summary which is important.

"Absence of Germinal Centers and Loss of Bcl-6+ Germinal Center B Cells but Preservation of AID+ B Cells in Lymph Nodes Early in COVID-19 Disease. We have used a human-tissue-imaging platform with quantitative high-resolution automated slide-scanning microscopy, exploiting both regular and confocal approaches and multispectral imaging, in order to interrogate human lymphoid and non-lymphoid organs at the single-cell level. These approaches crucially preserve architecture over broad swaths of tissue. Thoracic lymph nodes in severely ill COVID-19 patients who succumbed in less than 8 days after admission, less than 10 days from the onset of respiratory symptoms displayed a lack of germinal centers, and these were also absent in those who succumbed later, 15–36 days after admission."

"These data suggest an underlying basis for the lower quality and lack of durability of humoral immune responses observed during natural infection with SARS-CoV-2 and have significant implications for expectations of herd immunity. "

මෙහෙම එකක් දැම්මා කියලා ගොඩක් අය කියවන් නැ බන්, එක්කෝ මේක එක්ක සිංහලෙන් ට්‍රාන්ස්ලේට් කරපු එකක් දැම්මා නම් හරි.

 

[Thunderhawk]

ඇන්ටිගේ බොඩිය බලන්න ආපු උන් මේ කොනට වරෙල්ලා......

 

[Gamane]

TFS

 

[imhotep]

මෙහෙම එකක් දැම්මා කියලා ගොඩක් අය කියවන් නැ බන්, එක්කෝ මේක එක්ක සිංහලෙන් ට්‍රාන්ස්ලේට් කරපු එකක් දැම්මා නම් හරි.

Thanks... You do have a point. Maybe someone could translate.